Methyl 4-(acetylamino)-2-methoxybenzoate is one of metoclopramide impurities. Methyl 4-(acetylamino)-2-methoxybenzoate can be used as an intermediate for the synthesis of Irritable bowel syndrome modulator anticancer agents and other compounds.

CH223191 is a ligand-selective antagonist of the aryl hydrocarbon (dioxin) receptor (AhR), a ligand-dependent transcription factor that produces a wide range of biological and toxic effects in many species and tissues.

23-O-Acetylshengmanol-3-O-D-Xyloside is a natural product which can be extracted from Actaea racemosa (Black cohosh). It is structurally similar to 23-O-Acetylshengmanol-3-O-α-L-Arabinoside, except that beta-D-xyloside is substituted in place of α-L-Arabinoside. 23-O-Acetylshengmanol-3-OD-Xyloside is a potent enhancer of GABA-induced chloride currents.

BMS-442606 is an S-enantiomer of 6-hydroxybuspirone and is a 5-HT1A partial agonist. BMS-442606 has advantage of being cleared more slowly from blood compared to the R-enantiomer, but R form showed higher affinity and selectivity for the 5HT1A receptor compared to the S-isomer. Because both isomers together with racemic form were well tolerated and was not found any advantage of one of the enantiomers of over the racemic form. Finally, racemic form was chosen for further clinical development.

SNC80 is a highly selective agonist of δ opioid receptor but also binds to μ-δ opioid receptor heteromers to produce antinociception in mice. SNC80 also acts as anti-depressant, elicits dopamine-related behaviors and enhances behavioral responses to psychostimulants. SNC80 is not used medically, because of producing convulsions at high doses, although it is a useful drug in scientific research.

L-Gulonic acid, a diastereomer of d-gluconic acid is the one of a metabolite in the urinate cycle, is a substrate of L-Gulonate 3-dehydrogenase (GDH). This enzyme catalyzes the NAD(+)-linked dehydrogenation of L-gulonate into dehydro-L-gulonate. L-Gulonic acid is also a metabolite of the D-glucuronic acid pathway. It was suggested, that the measurement of a spectrum of urinary D-glucuronic acid metabolites might provide a more reliable index for assessment of the induction of hepatic xenobiotic-metabolizing enzyme activities in man than the determination of urinary D-glucaric acid alone.

N-Methylquipazine is a tertiary amine analog of quipazine. It binds at 5-HT3 sites with an affinity similar to that of quipazine. N-methylquipazine has a low affinity for 5-HT1A, 5-HT1B, 5-HT2, a1, a2 and muscarinic receptors. It produces a concentration-dependent increase in extracellular dopamine levels in the anterior medial prefrontal cortex. This action is dependent on the presence of Ca+2, is impulse-dependent, and depends on newly synthesized dopamine stores. The increase in the anterior medial prefrontal cortex dopamine levels by n-methylquipazine is probably, not mediated by its interaction with the 5-HT3 receptor. It might be an be an attractive candidate as a radioligand for the PET studies of 5-HT3 receptors in man.

Rivastigmine, (R)- is an impurity of a brain selective acetylcholinesterase inhibitor Rivastigmine. R-Rivastigmine enantiomer is pharmacologically less potent than S-enantiomer, which is used for the treatment of mild to moderate dementia of the Alzheimer's type and dementia due to Parkinson's disease.

ZINC 2,4,5-TRICHLOROPHENATE is a microbiocide, seed fungicide.

Triticonazole is a triazole pesticide/fungicide with broad antifungal activity. Like many azole fungicides, triticonazole likely inhibits 14-α demethylase, inhibiting ergosterol production and fungal cell wall synthesis. Additionally, triticonazole may inhibit aromatase.