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propoxycaine
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There is one exact (name or code) match for propoxycaine
Status:
US Previously Marketed
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
(1952)
Source URL:
First approved in 1952
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
Source URL:
Class (Stereo):
CHEMICAL (ACHIRAL)
Conditions:
Propoxycaine hydrochloride is a local anesthetic of the ester type that has a rapid onset of action and a longer duration of action than procaine hydrochloride. Propoxycaine Hydrochloride is the hydrochloride salt form of propoxycaine, a para-aminobenzoic acid ester. Propoxycaine binds to and inhibits voltage-gated sodium channels, thereby inhibiting the ionic flux required for the initiation and conduction of impulses. This results in a loss of sensation.
Showing 1 - 2 of 2 results
Status:
US Previously Marketed
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
(1952)
Source URL:
First approved in 1952
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
Source URL:
Class (Stereo):
CHEMICAL (ACHIRAL)
Conditions:
Propoxycaine hydrochloride is a local anesthetic of the ester type that has a rapid onset of action and a longer duration of action than procaine hydrochloride. Propoxycaine Hydrochloride is the hydrochloride salt form of propoxycaine, a para-aminobenzoic acid ester. Propoxycaine binds to and inhibits voltage-gated sodium channels, thereby inhibiting the ionic flux required for the initiation and conduction of impulses. This results in a loss of sensation.
Status:
US Previously Marketed
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
(1952)
Source URL:
First approved in 1952
Source:
RAVOCAINE AND NOVOCAIN W/ LEVOPHED by EASTMAN KODAK
Source URL:
Class (Stereo):
CHEMICAL (ACHIRAL)
Conditions:
Propoxycaine hydrochloride is a local anesthetic of the ester type that has a rapid onset of action and a longer duration of action than procaine hydrochloride. Propoxycaine Hydrochloride is the hydrochloride salt form of propoxycaine, a para-aminobenzoic acid ester. Propoxycaine binds to and inhibits voltage-gated sodium channels, thereby inhibiting the ionic flux required for the initiation and conduction of impulses. This results in a loss of sensation.