Nickel nitrate, a water soluble salt of Ni is irritating to the eyes, skin and, upon inhalation of the dust, respiratory tract. It may cause skin allergy. Nickel nitrate is a carcinogen, along with most other nickel compounds. The nickel ion is also toxic to aquatic organisms.

There is no information about biological and pharmacological application of lead stearate. But is known, that this is a toxic substance which can accumulate inside organism, because it can be absorbed through the skin.

There is no information related to biological or pharmacological application of lead(II) bromide (a combustion product of the gasoline additives lead (IV) tetraethyl and 1,2-dibromoethane). It is only known, that this substance possesses mutagenicity.

Stannous 2-ethylhexanoate is the stannous salt of 2-ehylhexanoic acid. It is used in the chemical industry as a catalyst for ring-opening polymerization of cyclic diesters in the production of polylactic acid and in the production of urethane foams, silicon, and other polymers.

Cobaltous carbonate is used as a feed additive for ruminants, horses and rabbits (dietary cobalt is needed for vitamin B12 synthesis). Cobaltous carbonate has carcinogen, mutagen and reproduction toxicant properties. Cobaltous carbonate is a skin and eye irritant, and a dermal and respiratory sensitizer. Its dust is a hazard to persons handling the substance. Exposure by inhalation must be avoided.

Lead chromate (previously known as chrome yellow), an insoluble chromium metal salt that widely used as a pigment in the production of paints, coatings, and plastics. Lead chromate possessed tumorigenic, genotoxic and clastogenic properties and was substituted by another pigment, cadmium yellow.

Pyrethrins are natural insecticides derived from chrysanthemum flowers containing a mixture of six components: pyrethrin I, cinerin I, jasmolin I, pyrethrin II, cinerin II, and jasmolin II. Pyrethrins induce a toxic effect in insects when they penetrate the cuticle and reach the nervous system. Pyrethrins bind to sodium channels that occur along the length of nerve cells. Sodium channels are responsible for nerve signal transmission along the length of the nerve cell by permitting the flux of sodium ions. When pyrethrins bind to sodium channels, normal function of the channels is obstructed thereby resulting in hyperexcitation of the nerve cell and, consequently, a loss of function of the nerve cell. The shutdown of the insect nervous system and death are most often the consequences of insect exposure to pyrethrins. Pyrethrin II has strong activity against Plasmodium falciparum.